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Reciprocal and Complementary Role of MET Amplification and EGFR T790M Mutation in Acquired Resistance to Kinase Inhibitors in Lung Cancer | Clinical Cancer Research
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MET/HGF pathway activation as a paradigm of resistance to targeted therapies - Ko - Annals of Translational Medicine
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Non-small cell lung cancer-small cell lung cancer transformation as mechanism of resistance to tyrosine kinase inhibitors in lung cancer
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The promise of selective MET inhibitors in non-small cell lung cancer with MET exon 14 skipping - Cancer Treatment Reviews
![Introduction - Targeting METex14 NSCLC - Text Module - MET Inhibition in NSCLC - Oncology - Clinical Care Options Introduction - Targeting METex14 NSCLC - Text Module - MET Inhibition in NSCLC - Oncology - Clinical Care Options](https://www.clinicaloptions.com/-/media/oncology/programs/nsclc-metex14/module-thumbs/cco-metex14-asco-cf_thumb2.png?rev=5b312d1500fb430198a84dee8244cc96)
Introduction - Targeting METex14 NSCLC - Text Module - MET Inhibition in NSCLC - Oncology - Clinical Care Options
![MET Oncogene in Non-Small Cell Lung Cancer: Mechanism of MET Dysregulation and Agents Targeting the HGF/c-Met Axis. - Abstract - Europe PMC MET Oncogene in Non-Small Cell Lung Cancer: Mechanism of MET Dysregulation and Agents Targeting the HGF/c-Met Axis. - Abstract - Europe PMC](https://europepmc.org/articles/PMC7104217/bin/OTT-13-2491-g0001.jpg)
MET Oncogene in Non-Small Cell Lung Cancer: Mechanism of MET Dysregulation and Agents Targeting the HGF/c-Met Axis. - Abstract - Europe PMC
![Early Alectinib Resistance From MET Amplification in ALK Rearranged NSCLC: Response to Crizotinib with Re-response to Alectinib and Crizotinib - Clinical Lung Cancer Early Alectinib Resistance From MET Amplification in ALK Rearranged NSCLC: Response to Crizotinib with Re-response to Alectinib and Crizotinib - Clinical Lung Cancer](https://els-jbs-prod-cdn.jbs.elsevierhealth.com/cms/asset/beee2da3-6560-43b5-ab65-92e14a762fb0/gr1.jpg)
Early Alectinib Resistance From MET Amplification in ALK Rearranged NSCLC: Response to Crizotinib with Re-response to Alectinib and Crizotinib - Clinical Lung Cancer
![MET amplification results in heterogeneous responses to osimertinib in EGFR-mutant lung cancer treated with erlotinib.,Cancer Science - X-MOL MET amplification results in heterogeneous responses to osimertinib in EGFR-mutant lung cancer treated with erlotinib.,Cancer Science - X-MOL](https://xpic.x-mol.com/20201008%2F10.1111_cas.14593.jpg)
MET amplification results in heterogeneous responses to osimertinib in EGFR-mutant lung cancer treated with erlotinib.,Cancer Science - X-MOL
![Reciprocal relationship between MET amplification and T790M mutation.... | Download Scientific Diagram Reciprocal relationship between MET amplification and T790M mutation.... | Download Scientific Diagram](https://www.researchgate.net/profile/Isao-Murakami/publication/47729355/figure/fig2/AS:394319564427276@1471024518957/Reciprocal-relationship-between-MET-amplification-and-T790M-mutation-A-tumor-numbers.png)
Reciprocal relationship between MET amplification and T790M mutation.... | Download Scientific Diagram
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MET inhibitors for targeted therapy of EGFR TKI-resistant lung cancer | Journal of Hematology & Oncology | Full Text
![EGFR pathway in NSCLC. Mutations, amplification or overexpression of... | Download Scientific Diagram EGFR pathway in NSCLC. Mutations, amplification or overexpression of... | Download Scientific Diagram](https://www.researchgate.net/profile/Claudia-Gonzalez-De-La-Rosa/publication/234595373/figure/fig2/AS:299687883100169@1448462567936/EGFR-pathway-in-NSCLC-Mutations-amplification-or-overexpression-of-growth-factors.png)
EGFR pathway in NSCLC. Mutations, amplification or overexpression of... | Download Scientific Diagram
![Figure 6 from Targeting Adenine Nucleotide Translocase-2 (ANT2) to Overcome Resistance to Epidermal Growth Factor Receptor Tyrosine Kinase Inhibitor in Non–Small Cell Lung Cancer | Semantic Scholar Figure 6 from Targeting Adenine Nucleotide Translocase-2 (ANT2) to Overcome Resistance to Epidermal Growth Factor Receptor Tyrosine Kinase Inhibitor in Non–Small Cell Lung Cancer | Semantic Scholar](https://d3i71xaburhd42.cloudfront.net/d6e96f49dbda7193a0c49ec86208afff21fd5b08/8-Figure6-1.png)
Figure 6 from Targeting Adenine Nucleotide Translocase-2 (ANT2) to Overcome Resistance to Epidermal Growth Factor Receptor Tyrosine Kinase Inhibitor in Non–Small Cell Lung Cancer | Semantic Scholar
![Chidamide increases the sensitivity of non-small cell lung cancer to crizotinib by decreasing c-MET mRNA methylation | bioRxiv Chidamide increases the sensitivity of non-small cell lung cancer to crizotinib by decreasing c-MET mRNA methylation | bioRxiv](https://www.biorxiv.org/content/biorxiv/early/2020/03/30/2020.03.28.012971/F1.large.jpg)
Chidamide increases the sensitivity of non-small cell lung cancer to crizotinib by decreasing c-MET mRNA methylation | bioRxiv
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MET inhibitors for targeted therapy of EGFR TKI-resistant lung cancer | Journal of Hematology & Oncology | Full Text
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The race to target MET exon 14 skipping alterations in non-small cell lung cancer: The Why, the How, the Who, the Unknown, and the Inevitable - ScienceDirect
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MET increased gene copy number and primary resistance to gefitinib therapy in non-small-cell lung cancer patients - Annals of Oncology
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